Rheumatoid arthritis (RA) is a chronic autoimmune disease involving persistent inflammation of multiple synovial joints, which results in progressive tissue destruction of bone and cartilage. A variety of innate and adaptive effector cells, including neutrophils, mastocytes , T and B lymphocytes, and monocytes/macrophages, are involved in the development of inflammation. Activation of these cells leads to the production of cytokines and mediators responsible for inflammation. Among these leukocytes, monocytes/macrophages and T cells are regarded as important players in the pathogenesis of RA. Monocytes/macrophages, which are central to the pathophysiology of inflammation, have been found to be activated in RA and produce tumor necrosis factor (TNF)-x, which is important in initiating, propagation and maintaining synovial inflammation.
(excerpt from: Inflammation Research (IR), Original Research Paper; "Decreased CD200R expression on monocyte-derived macrophages correlated with Th17/Treg imbalance and disease activity in rheumatoid arthritis patients" - ISSN: 1023-3830 (Print) 1420-908X (Online).)
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